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As the primary organ responsible for steroid metabolism, the liver is particularly susceptible to the toxic effects of 17--alkylated oral anabolicandrogenic steroids, such as stanozolol and methyltestosterone ( Mechanistic studies suggest that anabolicandrogenic steroids impair mitochondrial -oxidation and deplete intracellular glutathione stores, resulting in lipid peroxidation and activation of apoptotic signalling pathways

A perspective on the mller cell-neuron metabolic partnership in the inner retina

To further simplify data analysis we implemented two essential features in our assay

Recent studies have also shown that hyperhomocysteinemia compromises the autophagic capacity of Stx17

Research indicates that levels are often highest during sleep, as the body shifts into "repair and detox" mode