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Structure, biochemistry, and assembly of epithelial tight junctions

Treatment with 1-28 fragment significantly decreased tumor volume and weight in subcutaneous mouse models

Even after exposure ends, MC is still accessible to the liver thanks to slow release from other tissues including adipose

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The first hit involves dysregulated lipid metabolism leading to hepatocellular triglyceride accumulation, whereas the second hit encompasses oxidative stress and an amplified inflammatory response mediated by cytokines such as interleukins, transforming growth factor-1 (TGF-1), nuclear factor kappa B (NF-B), and tumor necrosis factor- (TNF-), ultimately predisposing to metabolic dysfunction-associated steatohepatitis (MASH), fibrosis, and cirrhosis 3