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The reduced activity of anorexigenic hormones such as glucagon-like peptide-1 (GLP-1), peptide tyrosine-tyrosine (PYY) and cholecystokinin has been linked to increased food consumption in patients with obesity (21)

It increases the working efficiency of mitochondria, which has an important role in the cell

Two collagen types dominate: Type I collagen (mature tendon structure) Type III collagen (early repair phase) In rodent studies, BPC-157-treated tendons showed: Increased type I collagen deposition Improved fiber parallel alignment Better biomechanical strength This suggests influence over: TGF- signalling Matrix metalloproteinases (MMPs) Extracellular matrix remodelling enzymes Balanced ECM remodelling prevents disorganized scar tissue

Most of the disorders affect the genes responsible for the bodys ability to produce collagen, a protein important for maintaining tissue structure

The proposal is straightforward: Move toward biomarker-driven patient stratification Design more flexible, biology-informed clinical trials Align regulatory approaches with what already works in oncology and dementia Dementia offers a useful reference point