guillain barre syndrome and glutathione Guillain‐Barré syndrome: a comprehensive review Guillain–Barré syndrome, transverse myelitis and

This may be particularly important if drugs promoting OPC differentiation lead to eventual depletion of the progenitor pool, and considering that aged OPCs have decreased capacity to mature and remyelinate, and are less responsive to pro-remyelinating drugs 101

Pyridoxine-dependent epilepsy and antiquitin deficiency resulting in neonatal-onset refractory seizures

Multi-omics integration was conducted to reveal core regulatory networks, and mechanistic validation focused on the expression of Nrf2, HO-1, and GPX4

Conversely, hypomethylation of CHGA and LINGO1 in the prefrontal cortex is linked to increased expression and abnormalities in neuroendocrine function, myelination, and neurodevelopment (Ueda et al., 2021)