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Catalase's capacity to lower oxidative stress and inflammation shows its potential as a therapeutic agent in the treatment of CAP, which might lead to better clinical results and reduced disease severity [95, 96]

That said, this method may overlook important aspects of biological complexity, such as variations in protein expression

These reactive species can damage surrounding tissues and DNA, promoting mutations and genomic instability

In: Tarr M

In our recent study, in addition to elucidating the pathogenic mechanism of FUS-associated ALS in both Drosophila and animal systems, we found that overexpression of GstO2, a Drosophila homolog of human GSTO1, reduces cytoplasmic FUS aggregates and attenuates neurodegenerative phenotypes, including mitochondrial dysfunction and neuronal toxicity 148