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However, most of this evidence comes from case reports and small observational cohorts, with a conspicuous lack of large-scale prospective trials

Furthermore, we confirm that the initial glutathionylation of the glutaredoxin moiety and not the glutathione transfer to the roGFP2 moiety is rate-limiting for the oxidation of fusion constructs at physiological or low micromolar GSSG concentrations, thus allowing glutaredoxin structure-function analysis in vivo

Research shows kidney cells actually depend on adequate glutathione for their own antioxidant protection

The PCA was weighted by group size

The central role of glutathione in the pathophysiology of human diseases