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Discussion The results of the present study showed that serosal application of acetic acid to induce gastric ulcer has resulted in gastric damage with elevated levels of pro-inflammatory cytokines along with increased neutrophil infiltration, lipid peroxidation, cyclooxygenases and prostaglandins, in conjunction with depleted antioxidant GSH in the gastric tissue

TB-500 reproduces the active region (the sequence Ac-LKKTETQ) of Thymosin Beta-4 , a protein tied to actin regulation, cell migration and wound healing

The detailed pharmacological therapeutic strategies are shown in Table 1

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