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Key DAMPs such as HMGB1 and ATP activate PRRs, including TLR4 and receptor for advanced glycation end products (RAGE), on macrophages, neutrophils, and fibroblasts

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It also modulated nuclear factor kappa B (NF-B) and transforming growth factor-beta (TGF-) signaling and reduced collagen type I (Col-I) and collagen type III (Col-III), connective tissue growth factor (CTGF), and neutrophil elastase (NE)