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Mao found that treating cancer cells with GPX4 inhibitors resulted in acute depletion of N-carbamyl-L-aspartate (C-ASP, a pyrimidine biosynthesis intermediate), accompanied by accumulation of uridine, which could be inhibited by fer-1, a ferroptosis inhibitor [18]

Figure 2B shows the proliferation of drug sensitive and resistant cells in the presence of increasing concentrations of tamoxifen

Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner