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Collectively, these results documented that the interference with Ea-domain glycosylation resulted in a dramatic decrease of intracellular proIGF-1Ea level and hence proIGF-1Ea and mature IGF-1 secretion

Niacin-responsive genetic disorders Congenital NAD deficiency-related disorders can result from mutations in genes involved in the uptake and transport of the various dietary NAD + precursors or in the distinct metabolic pathways leading to NAD + production (see Metabolism)