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The c-MET has been identified as an oncogene with convincing evidence, demonstrating the direct key roles of activating c-MET mutations and met amplification in promoting tumorigenesis in vivo (Graveel et al, 2004, 2005)

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It is also of clinical benefit to some patients with -thalassemia intermedia and it reduces the need for transfusions in a subset of individuals with -thalassemia major [46], [47], [48], [49], [50], [51], [52], [53]

Curran, S